This report describes the development of non-sustained ventricular tachycardia (NSVT) after a woman in her mid 50s underwent electroconvulsive therapy (ECT) for severe depression. Based on a retrospective study, we considered the NSVT in this patient to have been caused by takotsubo cardiomyopathy associated with ECT. Several reports have described VT after ECT, but the etiology remains unclear. However, our findings suggest that VT after ECT is associated with takotsubo cardiomyopathy. Here, we describe the progress of our patient, the findings of published reports of ECT-associated takotsubo cardiomyopathy, and emphasize the importance of frequent electrocardiography (ECG) for the early detection of asymptomatic takotsubo cardiomyopathy.
 Our patient was a woman in her mid 50s who had undergone surgery to treat contractile pericarditis six months previously. Four months thereafter, her thinking became clouded, and she began to blame herself constantly for different matters. She gradually exhibited marked psychomotor retardation over the next month. Two weeks before admission, she became unable to eat and had difficulty conversing. Her family accompanied her to our hospital.
 She was diagnosed with severe depression upon admission, and mirtazapine was started but immediately discontinued due to liver dysfunction. As her heart function was normal at that time, electroconvulsive therapy (ECT) was administered to treat depression. Thereafter, her symptoms gradually improved, but she developed ventricular tachycardia immediately after the fourth ECT session while in the operating room. Her sinus rhythm was restored after approximately 20 seconds. She recovered full consciousness after awakening from general anesthesia. However, the ECG on the following day revealed a prolonged QT interval and T-wave inversion; therefore, we consulted with cardiology. Several assessments ruled out myocardial infarction, but echocardiography demonstrated hypokinesia in the left ventricular apex. This suggested takotsubo cardiomyopathy. She had no subjective symptoms during this period. The left ventricular wall motion abnormality resolved after 12 days and she was discharged on hospital day 61 with stable mental status.
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